There is a substantial overlapin symptoms between the two conditions (reviewed byAarsland et al. 2004a, 2004b), probably reflecting a com-mon underlying cortical molecular pathology, with corti-cal LBs and more diffuse ?-synuclein pathology as com-mon diagnostic features of both conditions at autopsy.More specifically, several studies have suggested that,in many cortical regions, the amount of LB pathologydoes not differentiate DLB from PDD or PD (Hardingand Halliday, 2001; Tsuboi and Dixon, 2005).

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